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The production of molecules with adhesive properties which specifically recognize components of the extracellular matrix is a hallmark of cancer cells. This specificity for a given tissue suggests that the molecules involved also contribute to the invasive properties of cancer cells. They do so by regulating cell-cell and cell-matrix interactions. We show here that one adhesion molecule, TIMP-2, implicated in tumor invasion, stimulates the production of plasminogen activator inhibitor-1 (PAI-1), and acts as an inhibitor of urokinase-type plasminogen activator (u-PA). Through the induction of PAI-1, TIMP-2 appears to act as a promitogenic factor: it potentiates the ability of growth factors to promote the proliferation of fibroblasts and epithelial cells. In addition, it promotes the migration of epithelial cells. More recently, we have shown that TIMP-2 is up-regulated in fibroblasts after exposure to conditioned medium from fibroblasts that have been transformed by the SV40 large T antigen. Thus, TIMP-2 appears to be a component of a two-way interaction that provides the transformed epithelial cells with a growth advantage.Perioperative management of patients with gastric carcinoma.
Gastric cancer remains one of the most common and lethal malignancies in the world. Surgical resection, with either a curative or palliative intent, is the mainstay of treatment. Although this approach has largely eliminated the perioperative mortality and morbidity of an aggressive surgical approach, morbidity and mortality still occur in patients who do not undergo curative resection. Perioperative interventions have been recently performed for these patients in an attempt to improve the outcome, such as pre- and postoperative chemotherapy, pre- and postoperative radiotherapy, and growth factor therapy. Preoperative chemotherapy can provide either direct cytotoxicity for tumor cells or blockade of angiogenesis, and postoperative chemotherapy can reduce the tumor burden. Postoperative radiotherapy can also prevent local recurrence. Perioperative growth factor therapy (i.e., recombinant growth factors) can support the growth of tumor cells, which have already survived the operation, and
